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CBMT-15. MET INHIBITION DRIVES PGC1A DEPENDENT METABOLIC REPROGRAMMING AND ELICITS UNIQUE METABOLIC VULNERABILITIES IN GLIOBLASTOMA

The receptor kinase, c-MET, has emerged as a target for glioblastoma therapy. However, treatment resistance evolves inevitably. By performing a global metabolite screen with metabolite set enrichment coupled with transcriptome and gene set enrichment analysis and proteomic screening, we have identif...

詳細記述

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書誌詳細
出版年:Neuro Oncol
主要な著者: Zhang, Yiru, Nguyen, Trang, Zhao, Junfei, Shang, Enyuan, Torrini, Consuelo, Canoll, Peter D, Karpel-Massler, Georg, Siegelin, Markus
フォーマット: Artigo
言語:Inglês
出版事項: Oxford University Press 2019
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC6847878/
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/neuonc/noz175.137
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