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Exacerbated Responses to Oxidative Stress by an Na(+)Load in Isolated Nerve Terminals: the Role of ATP Depletion and Rise of [Ca(2+)](i)
We have explored the consequences of a [Na(+)](i) load and oxidative stress in isolated nerve terminals. The Na(+) load was achieved by veratridine (5–40 μm), which allows Na(+) entry via a voltage-operated Na(+) channel, and oxidative stress was induced by hydrogen peroxide (0.1–0.5 mm). Remarkably...
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| Published in: | J Neurosci |
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| Main Authors: | , , , |
| Format: | Artigo |
| Language: | Inglês |
| Published: |
Society for Neuroscience
2000
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| Subjects: | |
| Online Access: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6772497/ https://ncbi.nlm.nih.gov/pubmed/10704483 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.20-06-02094.2000 |
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