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Cortical Synaptic Integration In Vivo Is Disrupted by Amyloid-β Plaques
The accumulation of amyloid-β protein into plaques is a characteristic feature of Alzheimer's disease. However, the contribution of amyloid-β plaques to neuronal dysfunction is unknown. We compared intracellular recordings from neocortical pyramidal neurons in vivo in APP-Sw (Tg2576 transgenic...
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| Izdano u: | J Neurosci |
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| Glavni autori: | , , , , , |
| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
Society for Neuroscience
2004
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| Teme: | |
| Online pristup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6729398/ https://ncbi.nlm.nih.gov/pubmed/15140924 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.0462-04.2004 |
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