Cortical Synaptic Integration In Vivo Is Disrupted by Amyloid-β Plaques

The accumulation of amyloid-β protein into plaques is a characteristic feature of Alzheimer's disease. However, the contribution of amyloid-β plaques to neuronal dysfunction is unknown. We compared intracellular recordings from neocortical pyramidal neurons in vivo in APP-Sw (Tg2576 transgenic...

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Detaylı Bibliyografya
Yayımlandı:J Neurosci
Asıl Yazarlar: Stern, Edward A., Bacskai, Brian J., Hickey, Gregory A., Attenello, Frank J., Lombardo, Julianne A., Hyman, Bradley T.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Society for Neuroscience 2004
Konular:
Brief Communications
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC6729398/
https://ncbi.nlm.nih.gov/pubmed/15140924
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.0462-04.2004
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