Intracellular Accumulation of Amyloid-β (Aβ) Protein Plays a Major Role in Aβ-Induced Alterations of Glutamatergic Synaptic Transmission and Plasticity

Intracellular accumulation of amyloid-β (Aβ) protein has been proposed as an early event in AD pathogenesis. In patients with mild cognitive impairment, intraneuronal Aβ immunoreactivity was found especially in brain regions critically involved in the cognitive deficits of AD. Although a large body...

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Detalhes bibliográficos
Publicado no:J Neurosci
Main Authors: Ripoli, Cristian, Cocco, Sara, Li Puma, Domenica D., Piacentini, Roberto, Mastrodonato, Alessia, Scala, Federico, Puzzo, Daniela, D'Ascenzo, Marcello, Grassi, Claudio
Formato: Artigo
Idioma:Inglês
Publicado em: Society for Neuroscience 2014
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Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6705320/
https://ncbi.nlm.nih.gov/pubmed/25232124
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.1201-14.2014
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