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Lack of Pathology in a Triple Transgenic Mouse Model of Alzheimer's Disease after Overexpression of the Anti-Apoptotic Protein Bcl-2

Alzheimer's disease (AD) is characterized by the accumulation of plaques containing β-amyloid (Aβ) and neurofibrillary tangles (NFTs) consisting of modified tau. Although Aβ deposition is thought to precede the formation of NFTs in AD, the molecular steps connecting these two pathologies is not...

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Detalhes bibliográficos
Publicado no:J Neurosci
Main Authors: Rohn, Troy T., Vyas, Veera, Hernandez-Estrada, Tatiana, Nichol, Kathryn E., Christie, Lori-Ann, Head, Elizabeth
Formato: Artigo
Idioma:Inglês
Publicado em: Society for Neuroscience 2008
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6670712/
https://ncbi.nlm.nih.gov/pubmed/18354008
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.5620-07.2008
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