Atlastin-mediated membrane tethering is critical for cargo mobility and exit from the endoplasmic reticulum

Endoplasmic reticulum (ER) membrane junctions are formed by the dynamin-like GTPase atlastin (ATL). Deletion of ATL results in long unbranched ER tubules in cells, and mutation of human ATL1 is linked to hereditary spastic paraplegia. Here, we demonstrate that COPII formation is drastically decrease...

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Publicat a:Proc Natl Acad Sci U S A
Autors principals: Niu, Liling, Ma, Tianji, Yang, Feng, Yan, Bing, Tang, Xiao, Yin, Haidi, Wu, Qian, Huang, Yan, Yao, Zhong-Ping, Wang, Jifeng, Guo, Yusong, Hu, Junjie
Format: Artigo
Idioma:Inglês
Publicat: National Academy of Sciences 2019
Matèries:
PNAS Plus
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC6628656/
https://ncbi.nlm.nih.gov/pubmed/31239341
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1908409116
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