Atlastin-mediated membrane tethering is critical for cargo mobility and exit from the endoplasmic reticulum

Endoplasmic reticulum (ER) membrane junctions are formed by the dynamin-like GTPase atlastin (ATL). Deletion of ATL results in long unbranched ER tubules in cells, and mutation of human ATL1 is linked to hereditary spastic paraplegia. Here, we demonstrate that COPII formation is drastically decrease...

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Detalles Bibliográficos
Publicado en:Proc Natl Acad Sci U S A
Autores principales: Niu, Liling, Ma, Tianji, Yang, Feng, Yan, Bing, Tang, Xiao, Yin, Haidi, Wu, Qian, Huang, Yan, Yao, Zhong-Ping, Wang, Jifeng, Guo, Yusong, Hu, Junjie
Formato: Artigo
Lenguaje:Inglês
Publicado: National Academy of Sciences 2019
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PNAS Plus
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC6628656/
https://ncbi.nlm.nih.gov/pubmed/31239341
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1908409116
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