Atlastin-mediated membrane tethering is critical for cargo mobility and exit from the endoplasmic reticulum

Endoplasmic reticulum (ER) membrane junctions are formed by the dynamin-like GTPase atlastin (ATL). Deletion of ATL results in long unbranched ER tubules in cells, and mutation of human ATL1 is linked to hereditary spastic paraplegia. Here, we demonstrate that COPII formation is drastically decrease...

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Bibliografske podrobnosti
izdano v:Proc Natl Acad Sci U S A
Main Authors: Niu, Liling, Ma, Tianji, Yang, Feng, Yan, Bing, Tang, Xiao, Yin, Haidi, Wu, Qian, Huang, Yan, Yao, Zhong-Ping, Wang, Jifeng, Guo, Yusong, Hu, Junjie
Format: Artigo
Jezik:Inglês
Izdano: National Academy of Sciences 2019
Teme:
PNAS Plus
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC6628656/
https://ncbi.nlm.nih.gov/pubmed/31239341
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1908409116
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