Atlastin-mediated membrane tethering is critical for cargo mobility and exit from the endoplasmic reticulum

Endoplasmic reticulum (ER) membrane junctions are formed by the dynamin-like GTPase atlastin (ATL). Deletion of ATL results in long unbranched ER tubules in cells, and mutation of human ATL1 is linked to hereditary spastic paraplegia. Here, we demonstrate that COPII formation is drastically decrease...

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Detalhes bibliográficos
Publicado no:Proc Natl Acad Sci U S A
Main Authors: Niu, Liling, Ma, Tianji, Yang, Feng, Yan, Bing, Tang, Xiao, Yin, Haidi, Wu, Qian, Huang, Yan, Yao, Zhong-Ping, Wang, Jifeng, Guo, Yusong, Hu, Junjie
Formato: Artigo
Idioma:Inglês
Publicado em: National Academy of Sciences 2019
Assuntos:
PNAS Plus
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6628656/
https://ncbi.nlm.nih.gov/pubmed/31239341
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1908409116
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