טוען...
Dynamic changes in histone deacetylases following kidney ischemia-reperfusion injury are critical for promoting proximal tubule proliferation
Deranged histone deacetylase (HDAC) activity causes uncontrolled proliferation, inflammation, fibrosis, and organ damage. It is unclear whether deranged HDAC activity results in acute kidney injury in the renal hypoperfusion model of bilateral ischemia-reperfusion injury (IRI) and whether in vivo in...
שמור ב:
| הוצא לאור ב: | Am J Physiol Renal Physiol |
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| Main Authors: | , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
American Physiological Society
2019
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6580243/ https://ncbi.nlm.nih.gov/pubmed/30810062 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.00499.2018 |
| תגים: |
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