Podocyte-Specific Sialylation-Deficient Mice Serve as a Model for Human FSGS

BACKGROUND: The etiology of steroid-resistant nephrotic syndrome, which manifests as FSGS, is not completely understood. Aberrant glycosylation is an often underestimated factor for pathologic processes, and structural changes in the glomerular endothelial glycocalyx have been correlated with models...

पूर्ण विवरण

में बचाया:
ग्रंथसूची विवरण
में प्रकाशित:J Am Soc Nephrol
मुख्य लेखकों: Niculovic, Kristina M., Blume, Linda, Wedekind, Henri, Kats, Elina, Albers, Iris, Groos, Stephanie, Abeln, Markus, Schmitz, Jessica, Beuke, Esther, Bräsen, Jan H., Melk, Anette, Schiffer, Mario, Weinhold, Birgit, Münster-Kühnel, Anja K.
स्वरूप: Artigo
भाषा:Inglês
प्रकाशित: American Society of Nephrology 2019
विषय:
Basic Research
ऑनलाइन पहुंच:https://ncbi.nlm.nih.gov/pmc/articles/PMC6551790/
https://ncbi.nlm.nih.gov/pubmed/31040189
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2018090951
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