Podocyte-Specific Sialylation-Deficient Mice Serve as a Model for Human FSGS

BACKGROUND: The etiology of steroid-resistant nephrotic syndrome, which manifests as FSGS, is not completely understood. Aberrant glycosylation is an often underestimated factor for pathologic processes, and structural changes in the glomerular endothelial glycocalyx have been correlated with models...

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Bibliografiska uppgifter
I publikationen:J Am Soc Nephrol
Huvudupphovsmän: Niculovic, Kristina M., Blume, Linda, Wedekind, Henri, Kats, Elina, Albers, Iris, Groos, Stephanie, Abeln, Markus, Schmitz, Jessica, Beuke, Esther, Bräsen, Jan H., Melk, Anette, Schiffer, Mario, Weinhold, Birgit, Münster-Kühnel, Anja K.
Materialtyp: Artigo
Språk:Inglês
Publicerad: American Society of Nephrology 2019
Ämnen:
Basic Research
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC6551790/
https://ncbi.nlm.nih.gov/pubmed/31040189
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1681/ASN.2018090951
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