Protective Effects of a New C-Jun N-terminal Kinase Inhibitor in the Model of Global Cerebral Ischemia in Rats

c-Jun N-terminal kinase (JNK) is activated by various brain insults and is implicated in neuronal injury triggered by reperfusion-induced oxidative stress. Some JNK inhibitors demonstrated neuroprotective potential in various models, including cerebral ischemia/reperfusion injury. The objective of t...

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Publicado en:Molecules
Autores principales: Plotnikov, Mark B., Chernysheva, Galina A., Aliev, Oleg I., Smol’iakova, Vera I., Fomina, Tatiana I., Osipenko, Anton N., Rydchenko, Victoria S., Anfinogenova, Yana J., Khlebnikov, Andrei I., Schepetkin, Igor A., Atochin, Dmitriy N.
Formato: Artigo
Lenguaje:Inglês
Publicado: MDPI 2019
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Article
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC6539151/
https://ncbi.nlm.nih.gov/pubmed/31058815
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/molecules24091722
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