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Protective Effects of a New C-Jun N-terminal Kinase Inhibitor in the Model of Global Cerebral Ischemia in Rats

c-Jun N-terminal kinase (JNK) is activated by various brain insults and is implicated in neuronal injury triggered by reperfusion-induced oxidative stress. Some JNK inhibitors demonstrated neuroprotective potential in various models, including cerebral ischemia/reperfusion injury. The objective of t...

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Detalhes bibliográficos
Publicado no:Molecules
Main Authors: Plotnikov, Mark B., Chernysheva, Galina A., Aliev, Oleg I., Smol’iakova, Vera I., Fomina, Tatiana I., Osipenko, Anton N., Rydchenko, Victoria S., Anfinogenova, Yana J., Khlebnikov, Andrei I., Schepetkin, Igor A., Atochin, Dmitriy N.
Formato: Artigo
Idioma:Inglês
Publicado em: MDPI 2019
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC6539151/
https://ncbi.nlm.nih.gov/pubmed/31058815
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/molecules24091722
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