Protective Effects of a New C-Jun N-terminal Kinase Inhibitor in the Model of Global Cerebral Ischemia in Rats

c-Jun N-terminal kinase (JNK) is activated by various brain insults and is implicated in neuronal injury triggered by reperfusion-induced oxidative stress. Some JNK inhibitors demonstrated neuroprotective potential in various models, including cerebral ischemia/reperfusion injury. The objective of t...

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Bibliographic Details
Published in:Molecules
Main Authors: Plotnikov, Mark B., Chernysheva, Galina A., Aliev, Oleg I., Smol’iakova, Vera I., Fomina, Tatiana I., Osipenko, Anton N., Rydchenko, Victoria S., Anfinogenova, Yana J., Khlebnikov, Andrei I., Schepetkin, Igor A., Atochin, Dmitriy N.
Format: Artigo
Language:Inglês
Published: MDPI 2019
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Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC6539151/
https://ncbi.nlm.nih.gov/pubmed/31058815
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3390/molecules24091722
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