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Nuclear Translocation of Calmodulin in Pathological Cardiac Hypertrophy Originates from Ryanodine Receptor bound Calmodulin
In cardiac myocytes Calmodulin (CaM) bound to the ryanodine receptor (RyR2) constitutes a large pool of total myocyte CaM, but the CaM-RyR2 affinity is reduced in pathological conditions. Knock-in mice expressing RyR2 unable to bind CaM also developed hypertrophy and early death. However, it is unkn...
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| Izdano u: | J Mol Cell Cardiol |
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| Glavni autori: | , , , , , , , , , , , , , , |
| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
2018
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| Teme: | |
| Online pristup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC6402808/ https://ncbi.nlm.nih.gov/pubmed/30359562 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2018.10.011 |
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