Brain-Permeant and -Impermeant Inhibitors of Fatty Acid Amide Hydrolase Synergize with the Opioid Analgesic Morphine to Suppress Chemotherapy-Induced Neuropathic Nociception Without Enhancing Effects of Morphine on Gastrointestinal Transit

Lignende værker

• The cannabinoid CB(2) receptor agonist LY2828360 synergizes with morphine to suppress neuropathic nociception and attenuates morphine reward and physical dependence
  af: Iyer, Vishakh, et al.
  Udgivet: (2020)
• Brain permeant and impermeant inhibitors of fatty-acid amide hydrolase suppress the development and maintenance of paclitaxel-induced neuropathic pain without producing tolerance or physical dependence in vivo and synergize with paclitaxel to reduce tumor cell line viability in vitro
  af: Slivicki, Richard A., et al.
  Udgivet: (2019)
• A pro-nociceptive phenotype unmasked in mice lacking fatty-acid amide hydrolase
  af: Carey, Lawrence M, et al.
  Udgivet: (2016)
• Positive Allosteric Modulation of CB(1) Cannabinoid Receptor Signaling Enhances Morphine Antinociception and Attenuates Morphine Tolerance Without Enhancing Morphine- Induced Dependence or Reward
  af: Slivicki, Richard A., et al.
  Udgivet: (2020)
• Approximating protein flexibility through dynamic pharmacophore models: application to fatty acid amide hydrolase (FAAH)
  af: Bowman, Anna L., et al.
  Udgivet: (2011)