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A hypermorphic Nfkbid allele contributes to impaired thymic deletion of autoreactive diabetogenic CD8(+) T-cells in NOD mice(,)

In both NOD mice and humans, the development of type 1 diabetes (T1D) is dependent in part on autoreactive CD8(+) T-cells recognizing pancreatic ß-cell peptides presented by often quite common MHC class I variants. Studies in NOD mice previously revealed the common H2-K(d) and/or H2-D(b) class I mol...

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Bibliografske podrobnosti
izdano v:	J Immunol
Main Authors:	Presa, Maximiliano, Racine, Jeremy J., Dwyer, Jennifer R., Lamont, Deanna J., Ratiu, Jeremy J., Sarsani, Vishal Kumar, Chen, Yi-Guang, Geurts, Aron, Schmitz, Ingo, Stearns, Timothy, Allocco, Jennifer, Chapman, Harold D., Serreze, David V.
Format:	Artigo
Jezik:	Inglês
Izdano:	2018
Teme:	Article
Online dostop:	https://ncbi.nlm.nih.gov/pmc/articles/PMC6143397/ https://ncbi.nlm.nih.gov/pubmed/30127089 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1800465
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A hypermorphic Nfkbid allele contributes to impaired thymic deletion of autoreactive diabetogenic CD8(+) T-cells in NOD mice(,)

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