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Mitochondrial Dysfunction Triggers Synaptic Deficits via Activation of p38 MAP Kinase Signaling in Differentiated Alzheimer’s Disease Trans-Mitochondrial Cybrid Cells

Loss of synapse and synaptic dysfunction contribute importantly to cognitive impairment in Alzheimer’s disease (AD). Mitochondrial dysfunction and oxidative stress are early pathological features in AD-affected brain. However, the effect of AD mitochondria on synaptogenesis remains to be determined....

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Bibliografski detalji
Izdano u:J Alzheimers Dis
Glavni autori: Yu, Qing, Du, Fang, Douglas, Justin T., Yu, Haiyang, Yan, Shirley ShiDu, Yan, Shi Fang
Format: Artigo
Jezik:Inglês
Izdano: 2017
Teme:
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5935489/
https://ncbi.nlm.nih.gov/pubmed/28598851
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3233/JAD-170283
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