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Early deficits in synaptic mitochondria in an Alzheimer's disease mouse model
Synaptic dysfunction and the loss of synapses are early pathological features of Alzheimer's disease (AD). Synapses are sites of high energy demand and extensive calcium fluctuations; accordingly, synaptic transmission requires high levels of ATP and constant calcium fluctuation. Thus, synaptic...
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| 主要な著者: | , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
National Academy of Sciences
2010
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC2972922/ https://ncbi.nlm.nih.gov/pubmed/20937894 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1073/pnas.1006586107 |
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