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p53 promotes its own polyubiquitination by enhancing the HDM2 and HDMX interaction

HDM2 and HDMX are two homologs essential for controlling p53 tumor suppressor activity under normal conditions. Both proteins bind different sites on the p53 N‐terminus, and while HDM2 has E3 ubiquitin ligase activity towards p53, HDMX does not. Nevertheless, HDMX is required for p53 polyubiquitinat...

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Détails bibliographiques
Publié dans:Protein Sci
Auteurs principaux: Medina‐Medina, Ixaura, Martínez‐Sánchez, Mayra, Hernández‐Monge, Jesús, Fahraeus, Robin, Muller, Petr, Olivares‐Illana, Vanesa
Format: Artigo
Langue:Inglês
Publié: John Wiley and Sons Inc. 2018
Sujets:
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC5916125/
https://ncbi.nlm.nih.gov/pubmed/29524278
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/pro.3405
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