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Potent prion-like behaviors of pathogenic α-synuclein and evaluation of inactivation methods
The concept that abnormal protein aggregates show prion-like propagation between cells has been considered to explain the onset and progression of many neurodegenerative diseases. Indeed, both synthetic amyloid-like fibrils and pathogenic proteins extracted from patients’ brains induce self-template...
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| 出版年: | Acta Neuropathol Commun |
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| 主要な著者: | , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
BioMed Central
2018
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5907316/ https://ncbi.nlm.nih.gov/pubmed/29669601 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s40478-018-0532-2 |
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