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Potent prion-like behaviors of pathogenic α-synuclein and evaluation of inactivation methods

The concept that abnormal protein aggregates show prion-like propagation between cells has been considered to explain the onset and progression of many neurodegenerative diseases. Indeed, both synthetic amyloid-like fibrils and pathogenic proteins extracted from patients’ brains induce self-template...

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書目詳細資料
發表在:Acta Neuropathol Commun
Main Authors: Tarutani, Airi, Arai, Tetsuaki, Murayama, Shigeo, Hisanaga, Shin-ichi, Hasegawa, Masato
格式: Artigo
語言:Inglês
出版: BioMed Central 2018
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在線閱讀:https://ncbi.nlm.nih.gov/pmc/articles/PMC5907316/
https://ncbi.nlm.nih.gov/pubmed/29669601
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s40478-018-0532-2
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