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Increasing T‐type calcium channel activity by β‐adrenergic stimulation contributes to β‐adrenergic regulation of heart rates
KEY POINTS: Cav3.1 T‐type Ca(2+) channel current (I(Ca‐T)) contributes to heart rate genesis but is not known to contribute to heart rate regulation by the sympathetic/β‐adrenergic system (SAS). We show that the loss of Cav3.1 makes the beating rates of the heart in vivo and perfused hearts ex vivo,...
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| 出版年: | J Physiol |
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| 主要な著者: | , , , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
John Wiley and Sons Inc.
2018
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5878229/ https://ncbi.nlm.nih.gov/pubmed/29274077 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/JP274756 |
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