The convergent roles of NF-κB and ER stress in sunitinib-mediated expression of pro-tumorigenic cytokines and refractory phenotype in renal cell carcinoma

Lignende værker

• CRISPR/Cas9 genome-wide loss-of-function screening identifies druggable cellular factors involved in sunitinib resistance in renal cell carcinoma
  af: Makhov, Peter, et al.
  Udgivet: (2020)
• Depletion of intracellular zinc increases expression of tumorigenic cytokines VEGF, IL-6 and IL-8 in prostate cancer cells via NF-κB dependent pathway
  af: Golovine, Konstantin, et al.
  Udgivet: (2008)
• Are All Multi-targeted Tyrosine Kinase Inhibitors Created Equal? An In Vitro Study of Sunitinib and Pazopanib in Renal Cell Carcinoma Cell Lines
  af: Canter, Daniel, et al.
  Udgivet: (2011)
• Modulation of Akt/mTOR Signaling Overcomes Sunitinib Resistance in Renal and Prostate Cancer Cells
  af: Makhov, P. B., et al.
  Udgivet: (2012)
• Piperlongumine Inhibits NF-κB Activity and Attenuates Aggressive Growth Characteristics of Prostate Cancer Cells
  af: Ginzburg, Serge, et al.
  Udgivet: (2013)