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The Aβ oligomer eliminating D-enantiomeric peptide RD2 improves cognition without changing plaque pathology
While amyloid-β protein (Aβ) aggregation into insoluble plaques is one of the pathological hallmarks of Alzheimer’s disease (AD), soluble oligomeric Aβ has been hypothesized to be responsible for synapse damage, neurodegeneration, learning, and memory deficits in AD. Here, we investigate the in vitr...
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| Publicado no: | Sci Rep |
|---|---|
| Main Authors: | , , , , , , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Nature Publishing Group UK
2017
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5701182/ https://ncbi.nlm.nih.gov/pubmed/29176708 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41598-017-16565-1 |
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