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The heterotrimeric G protein Gβ(1) interacts with the catalytic subunit of protein phosphatase 1 and modulates G protein–coupled receptor signaling in platelets
Thrombosis is caused by the activation of platelets at the site of ruptured atherosclerotic plaques. This activation involves engagement of G protein–coupled receptors (GPCR) on platelets that promote their aggregation. Although it is known that protein kinases and phosphatases modulate GPCR signali...
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| Publicado no: | J Biol Chem |
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| Main Authors: | , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Society for Biochemistry and Molecular Biology
2017
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5555177/ https://ncbi.nlm.nih.gov/pubmed/28615442 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M117.796656 |
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