Smad1/5 is required for erythropoietin-mediated suppression of hepcidin in mice

Eitemau Tebyg

• Activin B Induces Noncanonical SMAD1/5/8 Signaling via BMP Type I Receptors in Hepatocytes: Evidence for a Role in Hepcidin Induction by Inflammation in Male Mice
  gan: Canali, Susanna, et al.
  Cyhoeddwyd: (2016)
• MicroRNA-130a Is Up-regulated in Mouse Liver by Iron Deficiency and Targets the Bone Morphogenetic Protein (BMP) Receptor ALK2 to Attenuate BMP Signaling and Hepcidin Transcription
  gan: Zumbrennen-Bullough, Kimberly B., et al.
  Cyhoeddwyd: (2014)
• Iron, erythropoietin, and inflammation regulate hepcidin in Bmp2-deficient mice, but serum iron fails to induce hepcidin in Bmp6-deficient mice
  gan: Wang, Chia-Yu, et al.
  Cyhoeddwyd: (2018)
• The SMAD Pathway Is Required for Hepcidin Response During Endoplasmic Reticulum Stress
  gan: Canali, Susanna, et al.
  Cyhoeddwyd: (2016)
• Limiting hepatic Bmp-Smad signaling by matriptase-2 is required for erythropoietin-mediated hepcidin suppression in mice
  gan: Nai, Antonella, et al.
  Cyhoeddwyd: (2016)