Mismatch repair proteins recruited to ultraviolet light-damaged sites lead to degradation of licensing factor Cdt1 in the G1 phase

Cdt1 is rapidly degraded by CRL4(Cdt2) E3 ubiquitin ligase after UV (UV) irradiation. Previous reports revealed that the nucleotide excision repair (NER) pathway is responsible for the rapid Cdt1-proteolysis. Here, we show that mismatch repair (MMR) proteins are also involved in the degradation of C...

Πλήρης περιγραφή

Αποθηκεύτηκε σε:
Λεπτομέρειες βιβλιογραφικής εγγραφής
Τόπος έκδοσης:Cell Cycle
Κύριοι συγγραφείς: Tanaka, Miyuki, Takahara, Michiyo, Nukina, Kohei, Hayashi, Akiyo, Sakai, Wataru, Sugasawa, Kaoru, Shiomi, Yasushi, Nishitani, Hideo
Μορφή: Artigo
Γλώσσα:Inglês
Έκδοση: Taylor & Francis 2017
Θέματα:
Report
Διαθέσιμο Online:https://ncbi.nlm.nih.gov/pmc/articles/PMC5397274/
https://ncbi.nlm.nih.gov/pubmed/28278049
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1080/15384101.2017.1295179
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