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Abnormal joint and bone wound healing in hemophilia mice is improved by extending factor IX activity after hemarthrosis

Wound healing requires interactions between coagulation, inflammation, angiogenesis, cellular migration, and proliferation. Healing in dermal wounds of hemophilia B mice is delayed when compared with hemostatically normal wild-type (WT) mice, with abnormal persistence of iron deposition, inflammatio...

Deskribapen osoa

Gorde:
Xehetasun bibliografikoak
Argitaratua izan da:Blood
Egile Nagusiak: Sun, Junjiang, Hua, Baolai, Livingston, Eric W., Taves, Sarah, Johansen, Peter B., Hoffman, Maureane, Ezban, Mirella, Monroe, Dougald M., Bateman, Ted A., Monahan, Paul E.
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: American Society of Hematology 2017
Gaiak:
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC5391623/
https://ncbi.nlm.nih.gov/pubmed/28039188
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2016-08-734053
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