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JNK potentiates TNF-stimulated necrosis by increasing the production of cytotoxic reactive oxygen species
The c-Jun NH(2)-terminal kinase (JNK) has been implicated in both cell death and survival responses to different stimuli. Here we reexamine the function of JNK in tumor necrosis factor (TNF)-stimulated cell death using fibroblasts isolated from wild-type, Mkk4(-/-) Mkk7(-/-), and Jnk1(-/-) Jnk2(-/-)...
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| Hlavní autoři: | , , , , |
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| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
Cold Spring Harbor Laboratory Press
2004
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC534651/ https://ncbi.nlm.nih.gov/pubmed/15545623 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1101/gad.1223004 |
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