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Bin1 and CD2AP polarise the endocytic generation of beta‐amyloid
The mechanisms driving pathological beta‐amyloid (Aβ) generation in late‐onset Alzheimer's disease (AD) are unclear. Two late‐onset AD risk factors, Bin1 and CD2AP, are regulators of endocytic trafficking, but it is unclear how their endocytic function regulates Aβ generation in neurons. We ide...
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| Gepubliceerd in: | EMBO Rep |
|---|---|
| Hoofdauteurs: | , , , , , , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
John Wiley and Sons Inc.
2016
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5210085/ https://ncbi.nlm.nih.gov/pubmed/27895104 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.15252/embr.201642738 |
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