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Extracellular K(+) rapidly controls NaCl cotransporter phosphorylation in the native distal convoluted tubule by Cl(−)‐dependent and independent mechanisms
KEY POINTS: High dietary potassium (K(+)) intake dephosphorylates and inactivates the NaCl cotransporter (NCC) in the renal distal convoluted tubule (DCT). Using several ex vivo models, we show that physiological changes in extracellular K(+), similar to those occurring after a K(+) rich diet, are s...
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| 出版年: | J Physiol |
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| 主要な著者: | , , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
John Wiley and Sons Inc.
2016
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5088235/ https://ncbi.nlm.nih.gov/pubmed/27457700 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/JP272504 |
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