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Amyloid-β(1–42) Aggregation Initiates Its Cellular Uptake and Cytotoxicity

The accumulation of amyloid β peptide(1–42) (Aβ(1–42)) in extracellular plaques is one of the pathological hallmarks of Alzheimer disease (AD). Several studies have suggested that cellular reuptake of Aβ(1–42) may be a crucial step in its cytotoxicity, but the uptake mechanism is not yet understood....

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Bibliografski detalji
Izdano u:J Biol Chem
Glavni autori: Jin, Sha, Kedia, Niraja, Illes-Toth, Eva, Haralampiev, Ivan, Prisner, Simon, Herrmann, Andreas, Wanker, Erich E., Bieschke, Jan
Format: Artigo
Jezik:Inglês
Izdano: American Society for Biochemistry and Molecular Biology 2016
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Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5016693/
https://ncbi.nlm.nih.gov/pubmed/27458018
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M115.691840
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