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Calorie seeking, but not hedonic response, contributes to hyperphagia in a mouse model for Prader–Willi syndrome
Prader–Willi syndrome (PWS) is a neurodevelopmental disorder caused by deletion or inactivation of paternally expressed imprinted genes on human chromosome 15q11‐q13, the most recognised feature of which is hyperphagia. This is thought to arise as a consequence of abnormalities in both the physiolog...
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| 出版年: | Eur J Neurosci |
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| 主要な著者: | , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
John Wiley and Sons Inc.
2015
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4949663/ https://ncbi.nlm.nih.gov/pubmed/26040449 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/ejn.12972 |
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