Calorie seeking, but not hedonic response, contributes to hyperphagia in a mouse model for Prader–Willi syndrome

Prader–Willi syndrome (PWS) is a neurodevelopmental disorder caused by deletion or inactivation of paternally expressed imprinted genes on human chromosome 15q11‐q13, the most recognised feature of which is hyperphagia. This is thought to arise as a consequence of abnormalities in both the physiolog...

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Veröffentlicht in:Eur J Neurosci
Hauptverfasser: Davies, Jennifer R., Humby, Trevor, Dwyer, Dominic M., Garfield, Alastair S., Furby, Hannah, Wilkinson, Lawrence S., Wells, Timothy, Isles, Anthony R.
Format: Artigo
Sprache:Inglês
Veröffentlicht: John Wiley and Sons Inc. 2015
Schlagworte:
Behavioural Neuroscience
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4949663/
https://ncbi.nlm.nih.gov/pubmed/26040449
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/ejn.12972
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