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PIAS1 regulates mutant Huntingtin accumulation and Huntington’s disease-associated phenotypes in vivo
The disruption of protein quality control networks is central to pathology in Huntington’s disease (HD) and other neurodegenerative disorders. The aberrant accumulation of insoluble high molecular weight protein complexes containing the Huntingtin (HTT) protein and SUMOylated protein corresponds to...
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| 出版年: | Neuron |
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| 主要な著者: | , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2016
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4942306/ https://ncbi.nlm.nih.gov/pubmed/27146268 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuron.2016.03.016 |
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