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Endothelin‐1 contributes to the progression of renal injury in sickle cell disease via reactive oxygen species
BACKGROUND AND PURPOSE: Endothelin‐1 (ET‐1) is increased in patients with sickle cell disease and may contribute to the development of sickle cell nephropathy. The current study was designed to determine whether ET‐1 acting via the ET(A) receptor contributes to renal injury in a mouse model of sickl...
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| 出版年: | Br J Pharmacol |
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| 主要な著者: | , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
John Wiley and Sons Inc.
2016
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4940621/ https://ncbi.nlm.nih.gov/pubmed/26561980 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/bph.13380 |
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