Androgen-sensitive hypertension associated with soluble guanylate cyclase-α(1) deficiency is mediated by 20-HETE

Dysregulated nitric oxide (NO) signaling contributes to the pathogenesis of hypertension, a prevalent and often sex-specific risk factor for cardiovascular disease. We previously reported that mice deficient in the α(1)-subunit of the NO receptor soluble guanylate cyclase (sGCα(1)(−/−) mice) display...

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Detaylı Bibliyografya
Yayımlandı:Am J Physiol Heart Circ Physiol
Asıl Yazarlar: Dordea, Ana C., Vandenwijngaert, Sara, Garcia, Victor, Tainsh, Robert E. T., Nathan, Daniel I., Allen, Kaitlin, Raher, Michael J., Tainsh, Laurel T., Zhang, Fan, Lieb, Wolfgang S., Mikelman, Sarah, Kirby, Andrew, Stevens, Christine, Thoonen, Robrecht, Hindle, Allyson G., Sips, Patrick Y., Falck, John R., Daly, Mark J., Brouckaert, Peter, Bloch, Kenneth D., Bloch, Donald B., Malhotra, Rajeev, Schwartzman, Michal L., Buys, Emmanuel S.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: American Physiological Society 2016
Konular:
Integrative Cardiovascular Physiology and Pathophysiology
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC4935526/
https://ncbi.nlm.nih.gov/pubmed/27199131
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00877.2015
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