Androgen-sensitive hypertension associated with soluble guanylate cyclase-α(1) deficiency is mediated by 20-HETE

Dysregulated nitric oxide (NO) signaling contributes to the pathogenesis of hypertension, a prevalent and often sex-specific risk factor for cardiovascular disease. We previously reported that mice deficient in the α(1)-subunit of the NO receptor soluble guanylate cyclase (sGCα(1)(−/−) mice) display...

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Argitaratua izan da:Am J Physiol Heart Circ Physiol
Egile Nagusiak: Dordea, Ana C., Vandenwijngaert, Sara, Garcia, Victor, Tainsh, Robert E. T., Nathan, Daniel I., Allen, Kaitlin, Raher, Michael J., Tainsh, Laurel T., Zhang, Fan, Lieb, Wolfgang S., Mikelman, Sarah, Kirby, Andrew, Stevens, Christine, Thoonen, Robrecht, Hindle, Allyson G., Sips, Patrick Y., Falck, John R., Daly, Mark J., Brouckaert, Peter, Bloch, Kenneth D., Bloch, Donald B., Malhotra, Rajeev, Schwartzman, Michal L., Buys, Emmanuel S.
Formatua: Artigo
Hizkuntza:Inglês
Argitaratua: American Physiological Society 2016
Gaiak:
Integrative Cardiovascular Physiology and Pathophysiology
Sarrera elektronikoa:https://ncbi.nlm.nih.gov/pmc/articles/PMC4935526/
https://ncbi.nlm.nih.gov/pubmed/27199131
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.00877.2015
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