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JNK mediates mouse liver injury through a novel Sab (SH3BP5) dependent pathway leading to inactivation of intramitochondrial Src
Sustained JNK activation has been implicated in many models of cell death and tissue injury. P-JNK interacts with the mitochondrial outer membrane protein, Sab (SH3BP5). Using knockdown or liver specific deletion of Sab we aimed to elucidate the consequences of this interaction on mitochondrial func...
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| I publikationen: | Hepatology |
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| Huvudupphovsmän: | , , , , |
| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
2016
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4874901/ https://ncbi.nlm.nih.gov/pubmed/26845758 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/hep.28486 |
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