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JNK mediates mouse liver injury through a novel Sab (SH3BP5) dependent pathway leading to inactivation of intramitochondrial Src
Sustained JNK activation has been implicated in many models of cell death and tissue injury. P-JNK interacts with the mitochondrial outer membrane protein, Sab (SH3BP5). Using knockdown or liver specific deletion of Sab we aimed to elucidate the consequences of this interaction on mitochondrial func...
Tallennettuna:
| Julkaisussa: | Hepatology |
|---|---|
| Päätekijät: | , , , , |
| Aineistotyyppi: | Artigo |
| Kieli: | Inglês |
| Julkaistu: |
2016
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| Aiheet: | |
| Linkit: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4874901/ https://ncbi.nlm.nih.gov/pubmed/26845758 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/hep.28486 |
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