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Tau-driven 26S proteasome impairment and cognitive dysfunction can be prevented early in disease by activating cAMP-PKA signaling

The ubiquitin proteasome system (UPS) degrades misfolded proteins including those implicated in neurodegenerative diseases. We investigated the effects of tau accumulation on proteasome function in a mouse model of tauopathy and in a cross to a UPS reporter mouse (line Ub-G76V-GFP). Accumulation of...

Täydet tiedot

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Bibliografiset tiedot
Julkaisussa:Nat Med
Päätekijät: Myeku, Natura, Clelland, Catherine L, Emrani, Sheina, Kukushkin, Nikolay V, Yu, Wai Haung, Goldberg, Alfred L, Duff, Karen E
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: 2015
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC4787271/
https://ncbi.nlm.nih.gov/pubmed/26692334
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/nm.4011
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