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Targeting the 26S Proteasome To Protect Against Proteotoxic Diseases
Aggregates of misfolded proteins can compromise the function of the 26S proteasome complex, leaving neurons susceptible to accelerated and impaired protein homeostasis, thereby contributing to the pathogenesis of neurodegeneration. Strategies aimed at enhancing the function of the 26S proteasome via...
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| Foilsithe in: | Trends Mol Med |
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| Main Authors: | , |
| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
2017
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5905406/ https://ncbi.nlm.nih.gov/pubmed/29233753 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.molmed.2017.11.006 |
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