Polydatin Protecting Kidneys against Hemorrhagic Shock-Induced Mitochondrial Dysfunction via SIRT1 Activation and p53 Deacetylation

Objectives. To ascertain if mitochondrial dysfunction (MD) of kidney cells is present in severe hemorrhagic shock and to investigate whether polydatin (PD) can attenuate MD and its protective mechanisms. Research Design and Methods. Renal tubular epithelial cells (RTECs) from rat kidneys experiencin...

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Detalhes bibliográficos
Publicado no:Oxid Med Cell Longev
Main Authors: Zeng, Zhenhua, Chen, Zhongqing, Xu, Siqi, Zhang, Qin, Wang, Xingmin, Gao, Youguang, Zhao, Ke-seng
Formato: Artigo
Idioma:Inglês
Publicado em: Hindawi Publishing Corporation 2016
Assuntos:
Research Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4783550/
https://ncbi.nlm.nih.gov/pubmed/27057271
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1155/2016/1737185
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