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β-arrestin drives MAP kinase signaling from clathrin-coated structures after GPCR dissociation
β-arrestins critically regulate G protein-coupled receptor (GPCR) signaling, not only 'arresting' the G protein signal but also modulating endocytosis and initiating a discrete G protein-independent signal via MAP kinase(1–3). Despite enormous recent progress toward understanding biophysic...
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| Publicado no: | Nat Cell Biol |
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| Main Authors: | , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2016
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4767649/ https://ncbi.nlm.nih.gov/pubmed/26829388 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/ncb3307 |
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