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Slx4 and Rtt107 control checkpoint signalling and DNA resection at double-strand breaks
The DNA damage checkpoint pathway is activated in response to DNA lesions and replication stress to preserve genome integrity. However, hyper-activation of this surveillance system is detrimental to the cell, because it might prevent cell cycle re-start after repair, which may also lead to senescenc...
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| 出版年: | Nucleic Acids Res |
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| 主要な著者: | , , , , , , , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
Oxford University Press
2016
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4737138/ https://ncbi.nlm.nih.gov/pubmed/26490958 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/nar/gkv1080 |
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