Mitochondrial Superoxide Contributes to Hippocampal Synaptic Dysfunction and Memory Deficits in Angelman Syndrome Model Mice

Lignende værker

• Genetic Reduction of the α1 Subunit of Na/K-ATPase Corrects Multiple Hippocampal Phenotypes in Angelman Syndrome
  af: Kaphzan, Hanoch, et al.
  Udgivet: (2013)
• Reversal of Impaired Hippocampal Long-term Potentiation and Contextual Fear Memory Deficits in Angelman Syndrome Model Mice by ErbB Inhibitors
  af: Kaphzan, Hanoch, et al.
  Udgivet: (2012)
• Amyloid β-Induced Impairments in Hippocampal Synaptic Plasticity Are Rescued by Decreasing Mitochondrial Superoxide
  af: Ma, Tao, et al.
  Udgivet: (2011)
• Synaptic dysfunction, memory deficits and hippocampal atrophy due to ablation of mitochondrial fission in adult forebrain neurons
  af: Oettinghaus, B, et al.
  Udgivet: (2016)
• Superoxide Anion-Dependent Mitochondrial Fission Contributes to Hippocampal Synaptic Dysfunction in Stress-Susceptible Mice
  af: Xiwei Li, et al.
  Udgivet: (2025-09-01)