The participation of insulin-like growth factor-binding protein 3 released by astrocytes in the pathology of Alzheimer’s disease

Lignende værker

• Continuation of Exercise Is Necessary to Inhibit High Fat Diet-Induced β-Amyloid Deposition and Memory Deficit in Amyloid Precursor Protein Transgenic Mice
  af: Maesako, Masato, et al.
  Udgivet: (2013)
• Exercise Is More Effective than Diet Control in Preventing High Fat Diet-induced β-Amyloid Deposition and Memory Deficit in Amyloid Precursor Protein Transgenic Mice
  af: Maesako, Masato, et al.
  Udgivet: (2012)
• Presenilin Regulates Insulin Signaling via a γ-Secretase-independent Mechanism
  af: Maesako, Masato, et al.
  Udgivet: (2011)
• N-cadherin enhances APP dimerization at the extracellular domain and modulates Aβ production
  af: Asada-Utsugi, Megumi, et al.
  Udgivet: (2011)
• N-cadherin Regulates p38 MAPK Signaling via Association with JNK-associated Leucine Zipper Protein: IMPLICATIONS FOR NEURODEGENERATION IN ALZHEIMER DISEASE
  af: Ando, Koichi, et al.
  Udgivet: (2011)