טוען...
Presenilin Regulates Insulin Signaling via a γ-Secretase-independent Mechanism
Presenilin (PS), a causative molecule of familial Alzheimer disease, acts as a crucial component of the γ-secretase complex, which is required to cleave type I transmembrane proteins such as amyloid precursor protein and Notch. However, it also functions through γ-secretase-independent pathways. Rec...
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| Main Authors: | , , , , , , , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
American Society for Biochemistry and Molecular Biology
2011
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| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3137102/ https://ncbi.nlm.nih.gov/pubmed/21622565 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M111.248922 |
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