Aggregated α-synuclein and complex I deficiency: exploration of their relationship in differentiated neurons

α-Synuclein becomes misfolded and aggregated upon damage by various factors, for example, by reactive oxygen species. These aggregated forms have been proposed to have differential toxicities and their interaction with mitochondria may cause dysfunction within this organelle that contributes to the...

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Bibliografische gegevens
Gepubliceerd in:Cell Death Dis
Hoofdauteurs: Reeve, A K, Ludtmann, M HR, Angelova, P R, Simcox, E M, Horrocks, M H, Klenerman, D, Gandhi, S, Turnbull, D M, Abramov, A Y
Formaat: Artigo
Taal:Inglês
Gepubliceerd in: Nature Publishing Group 2015
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Original Article
Online toegang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4650719/
https://ncbi.nlm.nih.gov/pubmed/26181201
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/cddis.2015.166
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