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Aggregated α-synuclein and complex I deficiency: exploration of their relationship in differentiated neurons

α-Synuclein becomes misfolded and aggregated upon damage by various factors, for example, by reactive oxygen species. These aggregated forms have been proposed to have differential toxicities and their interaction with mitochondria may cause dysfunction within this organelle that contributes to the...

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Библиографические подробности
Опубликовано в: :Cell Death Dis
Главные авторы: Reeve, A K, Ludtmann, M HR, Angelova, P R, Simcox, E M, Horrocks, M H, Klenerman, D, Gandhi, S, Turnbull, D M, Abramov, A Y
Формат: Artigo
Язык:Inglês
Опубликовано: Nature Publishing Group 2015
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Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC4650719/
https://ncbi.nlm.nih.gov/pubmed/26181201
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/cddis.2015.166
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