Aggregated α-synuclein and complex I deficiency: exploration of their relationship in differentiated neurons

α-Synuclein becomes misfolded and aggregated upon damage by various factors, for example, by reactive oxygen species. These aggregated forms have been proposed to have differential toxicities and their interaction with mitochondria may cause dysfunction within this organelle that contributes to the...

Полное описание

Сохранить в:
Библиографические подробности
Опубликовано в: :Cell Death Dis
Главные авторы: Reeve, A K, Ludtmann, M HR, Angelova, P R, Simcox, E M, Horrocks, M H, Klenerman, D, Gandhi, S, Turnbull, D M, Abramov, A Y
Формат: Artigo
Язык:Inglês
Опубликовано: Nature Publishing Group 2015
Предметы:
Original Article
Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC4650719/
https://ncbi.nlm.nih.gov/pubmed/26181201
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/cddis.2015.166
Метки: Добавить метку
Нет меток, Требуется 1-ая метка записи!

Схожие документы