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Aggregated α-synuclein and complex I deficiency: exploration of their relationship in differentiated neurons
α-Synuclein becomes misfolded and aggregated upon damage by various factors, for example, by reactive oxygen species. These aggregated forms have been proposed to have differential toxicities and their interaction with mitochondria may cause dysfunction within this organelle that contributes to the...
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| Publicado en: | Cell Death Dis |
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| Autores principales: | , , , , , , , , |
| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
Nature Publishing Group
2015
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4650719/ https://ncbi.nlm.nih.gov/pubmed/26181201 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/cddis.2015.166 |
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